RESUMO Ascaridíase por Ascaridia hermaphrodita é uma doença parasitária comum em papagaios-verdadeiros. Sua contaminação ocorre pela ingestão de ovos. O tráfico de aves silvestres pode facilitar a migração errática visceral desse parasita. Este relato descreve a migração errática de A. hermaphrodita e lesões associadas em papagaio-verdadeiro. Trata-se de um relato retrospectivo, analisado a partir de uma série de apreensões de aves no estado da Paraíba, Brasil. A necropsia revelou intensa infestação ascarídea intestinal. Também foram observadas lesões hepáticas em uma ave, associadas à migração do parasita. A estereoscopia permitiu distinguir a morfologia dos parasitas. Resumidamente, essa condição em aves silvestres é pouco conhecida e pode ser considerada, inclusive, associada à comercialização ilegal.

ABSTRACT Ascaridiasis by Ascaridia hermaphrodita is a common parasitic disease in blue-fronted Amazon parrots (Amazona aestiva) and the contamination occurs by ingestion of eggs. Illegal trade of wild birds can increase the frequency of visceral erratic migration by this parasite. Hence, this report describes the erratic migration of A. hermaphrodita and associated lesions in a blue-fronted Amazon parrot. This is a retrospective study, which originated from several apprehension of illegal birds in the state of Paraíba, Brazil. Necropsy revealed intense intestinal ascarid infestation. Hepatic lesions were also observed associated with the parasite migration. Stereoscopy allowed the identification of the morphology of the parasites. Summarizing, this condition in wild birds is not fully understood and may be associated with illegal trade.

We present here a hydrothermal synthesis on spherical shape molybdenum di-sulfide (MoS2) micro-particles using thiomolybdate salts and sodium silicate as reducing agent. To understand the role of cobalt promoters on this particular MoS2 spherical shape a second reaction was carried out using same precursors plus addition of Co following same pressure and temperature conditions. Both products (before and after Co promoter) were characterized using scanning electron and transmission electron microscopic analysis. From SEM measurements a spherical average size diameter of ~ 2.855 µm on pure MoS2 is observed and disperse surface once cobalt is incorporated into the reaction. From TEM observations an interlayer average distance of ~ 0.63 nm is obtained for MoS2-MoS2 slabs on samples with Co content. X-ray diffraction indicated principal crystallographic planes to be (002), (100), (101), (102), (103), (006), (105), and (110) for both MoS2 and MoS2/Co samples.

Presentamos aquí una síntesis química de micropartículas esféricas de sulfuro de molibdeno (MoS2) utilizando sales de tiomolibdato y silicato de sodio como agente reductivo. Para comprender el rol de los promotores de cobalto (Co) en estas particulares micro-esferas de MoS2, una segunda reacción fue realizada utilizando los mismos precursores y la adición de Cobalto bajo las mismas condiciones de presión y temperatura. Ambos productos (antes y después del Co) fueron caracterizados utilizando microscopios de barrido y transmisión electrónicos (SEM y TEM). Los resultados del SEM indican un diámetro promedio de ~2.855 µm en esferas de puro MoS2, así como una dispersión cuando el cobalto es incorporado en la reacción. Observaciones en TEM indican una distancia promedio de ~0.63 nm en las laminas de MoS2 para muestras que contienen cobalto. Los resultados de rayos-X indican que los principales planos de difraccion son: (002), (100), (101), (102), (103), (006), (105), y (110) para ambas muestras las de MoS2 y MoS2/Co.

OBJECTIVE: To evaluate the outcomes of acute myeloid leukemia patients who were older than 60 years of age at the time of diagnosis following the implementation of a treatment algorithm based on age, performance status, and cytogenetic results. METHODS: We retrospectively compared the results of 31 elderly acute myeloid leukemia patients (median age of 74 years) who were treated according to the new algorithm. RESULTS: Fifteen patients with a good performance status and no unfavorable karyotypes were treated with either intensive cytotoxic chemotherapy (<70 years, nine cases) or adapted etoposide, 6-thioguanine and idarubicine (>70 years, six cases); 16 cases with a poor performance status or unfavorable cytogenetics received supportive care only. Six patients achieved a complete remission and two achieved a partial remission after chemotherapy. There were three toxic deaths during induction, two in the adapted etoposide, 6-thioguanine and idarubicine group and one in the intensive cytotoxic chemotherapy group. The overall median survival time was 2.96 months, 1.3 months in the supportive care group, and 4.6 months in the treatment group. CONCLUSIONS: Our results illustrate the importance of treatment guidelines adapted to local resources in an attempt to improve the survival of elderly acute myeloid leukemia patients in developing countries.