Prenatally programmed hypertension: role of maternal diabetes. G.N. Gomes and F.Z. Gil.
Figure 3. Changes on vascular diameter of the mesenteric arterial bed. Maternal diabetes caused an impaired response to endothelium-dependent agents. Bar graphs show the response of the mesenteric microvessels to A, acetylcholine (300 µg/mL); B, bradykinin (3.0 µg/mL) and C, sodium nitroprusside (1.0 mg/mL) in control (C, open bars, N = 6) and diabetic offspring (DO, filled bars, N = 9) groups. Data are reported as means ± SEM of percent change. *P < 0.05 compared to control (analysis of variance followed by the Bonferroni test). Reprinted, with permission, from Rocha et al. (8).