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Progression of ventricular repolarization in left bundle branch block in non-compaction of the myocardium

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Progression of ventricular repolarization in left bundle branch block in non-compaction of the myocardium

Lurildo Ribeiro Saraiva; Giordano Bruno Parente; Ricardo Loureiro; Thiago B. Saraiva Leão

Universidade Federal de Pernambuco, Recife, PE, Brazil

Mailing Address Mailing Address: Lurildo R. Saraiva Av. Prof. Moraes Rego, SN Hospital das Clínicas UFPE, Disciplina de Cardiologia Cidade Universitária 52070-420 - Recife, PE, Brazil E-mail: lurildocleano@hotmail.com, lurildo@cardiol.br

Key words: Left bundle branch block, circumferential subendocardial ischemia, non-compacted myocardium.

Male, 34-years-old, developed a rapidly progressive heart failure four years ago, resulting from a left ventricular (LV) noncompaction cardiomyopathy according to findings from magnetic resonance angiography. The initial electrocardiogram (Fig. 1) showed a first-degree AV block, left ventricular and left atrial overload and left bundle branch block, with SÂT at +30°, and biphasic T waves in D1 and V6. Recently, after a viral infection of the respiratory tract he presented with a severe exertional limitation. Doppler echocardiogram (Fig. 2) demonstrated diffuse hypokinesia and a significant decrease in LV ejection fraction (0.38) with mild mitral regurgitation, final LV diastolic diameter of 6.3cm and final systolic diameter of 5.1 cm, in addition to detecting the characteristic trabeculations in the internal face of the septal-apical myocardium with intratrabecular blood flow. ECG (Figure 3) showed a higher degree of aberrant ventricular activation and significant T wave inversion in anterolateral and inferior walls, with a diametrically opposed SÂT at -145°.




Comments

The electrocardiographic concept of circumferential subendocardial ischemia because of SÂT displacement to the right upper quadrant in the three-vessel coronary artery disease or the like, indicative of a sudden increase in LV end-diastolic pressure, may be pertinent to this form of cardiomyopathy, for which information on ECG alterations is scarce.

Manuscript received November 15, 2005; revised Manuscript received November 21, 2005; accepted November 21, 2005.

  • Mailing Address:
    Lurildo R. Saraiva
    Av. Prof. Moraes Rego, SN
    Hospital das Clínicas UFPE, Disciplina de Cardiologia
    Cidade Universitária
    52070-420 - Recife, PE, Brazil
    E-mail:
  • Publication Dates

    • Publication in this collection
      07 Mar 2007
    • Date of issue
      Nov 2006
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    E-mail: revista@cardiol.br