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Journal of Venomous Animals and Toxins including Tropical Diseases

On-line version ISSN 1678-9199

J. Venom. Anim. Toxins incl. Trop. Dis vol.10 no.2 Botucatu  2004

http://dx.doi.org/10.1590/S1678-91992004000200009 

ERRATA

 

 

ERRATA V.9, N.1, 2003

Original Paper The Excitatory effects of Buthus C56 toxin on Drosophila larval neuromuscular junction. J. Venom. Anim. Toxins incl. Trop. Dis, v.9, 1, p.65-75, 2003.

In Discussion, p. 72, the last two sentences:

NMDA activation type of glutamate receptors at the glutaminergic synapses have been suggested as causing an opening of large number of calcium channels with consequent intracellular calcium excess involved in the excitotoxicity in degenerative diseases (14). When EJP amplitude was increased with concentration, maximum increase in EJP frequency in 0.4 mM Ca2+/4 mM Mg2+ Ringer was suggested to be due to maximum NMDA activation type of glutamate inotropic receptors for the augmented Ca2+ influx in C56 toxin-exposed Drosophila larval n-m junction.

Should read:

It was suggested that activation of NMDA type of glutamate receptors at the glutaminergic synapses resulted into opening of large number of calcium channels with consequent intracellular calcium excess involved in the excitotoxicity in degenerative diseases (14).

When amplitude of EJPs was increased dependently on concentration, maximum increase in EJP frequency in 0.4 mM Ca2+ / 4 mM Mg2+ Ringer was suggested to be due to maximum activation of NMDA type of glutamate inotropic receptors for the augmented Ca 2+ influx on toxin C56 exposed Drosophila larval n-m junction.

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